ハードウェアデバイスに変化の兆し？

現在、世界の主要なシミュレーターメーカーは、内視鏡手術用バーチャルリアリティシミュレーターに、自社ソフトウェアと共にImmersion社（米国）のハードウェアデバイス（ハンドル部分）を採用して販売しています。このハードデバイス部分ではほぼImmersion社の独占状態となっていました。

そこに、スイスのメーカーが、新製品を開発してきました。

詳細はこちら：http://www.xitact.com/products/lap/

とてもコンパクトで、Haptic(触覚）機能の有るタイプと無いタイプの両方あるようです。

シミュレーターを取り巻く環境の変化と更なる進化が期待されます。

Comments

[投稿： wohormelpermA]

MADISON, N.J., July 14, 2008 /PRNewswire-FirstCall/ -- Wyeth announced today that value Jamie D. Happas of the New Jersey ‚litist Court in New Brunswick has granted Wyeth's agitation for laconic judgment in two hormone remedial prospectus cases that were scheduled for examination later this year. The court dismissed the cases of Bailey v. Wyeth, whicdh was to enjoy begun on September 15, and DeBoard v. Wyeth, which had an October 27 trying out date. The plaintiffs in both cases claimed they come abouted tit cancer as a outcome of the use of Prempro(TM) and Premarin(R), hormone remedy results marketed by Wyeth, as fit
as Provera(R), a progestin goods marketed by Upjohn.

In granting the agitations, deem Happas ruled, surroudned by other things, that the plaintiffs "failed to state look after the limited discernmentification of show high-priority to defeat the conviction of adequacy afforded FDA-approved labeling" by New Jersey law and that the warnings were "average as a situation of law."

[投稿： yjnkst mkdlrieyo]

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[投稿： StWhite]

Hi, folks
For the latest study, it was
The most common diseases associated with are chronic viral hepatitis, alcoholism, and ( scarring of the liver ). Moreover, chronic viral hepatitis is common in alcoholism, and both and alcoholism cause cirrhosis which usually precedes the development of cancer. Therefore, the contributions and interrelationships of , viral hepatitis, and cirrhosis in the development of liver cancer are complex. Despite the complexity, it is important to try to understand the contributions of each disease so that patients at highest risk for liver cancer can be targeted for . Theoretically, they also might be targeted with treatments that prevent the development of liver cancer, when such treatments are developed.
A practical study published in October 2007 has extended important the latest about the homogeneity of liver cancer to chronic viral hepatitis, alcoholism, and cirrhosis. This is a manly study owing to it used the records of Swedish health registries to discern patients for inclusion in the study. The Swedish registries consist of cleverness on the entire society of Sweden. They are hefty and whole registries and dominion been in avail for multifarious age. In materiality, they posses provided a treasure of score about rife diseases.
An analysis of the Swedish data demonstrated that among patients with alcoholism slick was slightly more than a coupled accrual in the risk of liver cancer in that compared with the general human relay. This insufficient development suggests that alcoholism alone is not strongly related to the development of liver cancer. On the other hand, patients who were alcoholics but also developed cirrhosis, presumably since a settlement of their alcoholism, had a 22 - commune aggrandizement in the risk of liver cancer in that compared with the general general public. Remarkably, the development of cirrhosis in alcoholics substantially increases the risk for liver cancer.
Patients with chronic viral hepatitis had a 34 - district greater risk for liver cancer because compared with the general humans. Patients with both chronic viral hepatitis and cirrhosis, however, had a much greater burgeoning in the development of liver cancer - - 118 - district. ( Presumably, the cirrhosis was caused by the chronic hepatitis. ) Decidedly, the combination of chronic viral hepatitis and cirrhosis has a exact well-made association with the development of liver cancer. This association is much stronger than the association of the combination of alcoholism and cirrhosis with liver cancer. The stronger association with viral hepatitis than alcohol supports a greater role for the hepatitis viruses through compared with alcohol in promoting liver cancer.
What can we conclude from this study ? Primeval, the risk of liver cancer is remarkably fresh in patients with chronic viral hepatitis and cirrhosis. The risk also is another in patients who are alcoholic and own cirrhosis, although the risk is less. Second, if we craving to substantially prevent the development of liver cancer, we duty spot patients before they evolve cirrhosis and accordingly prevent cirrhosis. Third, existing and newer techniques for liver cancer surveillance probably should be applied to all patients with both chronic viral hepatitis and cirrhosis and possibly to patients with both alcoholism and cirrhosis. Fourth,we need to develop better techniques for liver cancer surveillance. A satisfactory solution to the problem of liver cancer in chronic viral hepatitis and alcoholism will not be quick or easy.
With all the best
Steven